Molecular Biology


  • Caradonna

    Sal J. Caradonna, PhD

    Professor and Chair
    Science Center 120
    856 566-6056

    My laboratory is interested in the post-translational mechanisms that regulate the proteins involved in base-excision repair of DNA. We are studying the aberrant pathways that lead to uracil misincorporation into DNA as well as the regulated cytosine deamination pathways of the APOBEC family of cytosine deaminases.

  • Katrina Cooper, PhD

    Katrina Cooper, PhD

    Associate Professor
    Science Center 362
    856 566-2887

    The long-term goal of my research is to elucidate the molecular mechanism by which cells both decide and execute their fates in response to environmental cues. Misinterpretation of these signals can result in the cell choosing the incorrect fate, which, in turn, can lead to neoplasia, i.e. tumor formation. The focus of my group centers on a highly conserved protein called cyclin C that is a newly identified tumor suppressor. Befitting this title, this nuclear protein plays an important role in pro-survival (autophagy) and pro-death decisions dependent upon the type of stress sensed.

  • Katrina Cooper, PhD

    Renée M. Demarest, PhD

    Assistant Professor
    856 566-6402
    Science Center 276

    The main goal of my lab is to delineate the molecular pathways required for T-cell acute lymphoblastic leukemia (T-ALL) formation, maintenance, and relapse. We are currently delineating the requirement for Notch, Ikaros, and myc in this process, and exploring novel therapeutic regimens for T-ALL treatment. In addition, we are developing novel methods to treat various murine models of cancer with human equivalent chemotherapeutic regimens in order to increase the translational success of new therapeutics to the clinic.

  • Ronald Ellis, PhD

    Ronald Ellis, PhD

    Science Center 316
    856 566-2768
    Fax: 856 566-6291

    Control of Germ Cell Fate: Animals must produce sperm or eggs to reproduce. Although these cell types differ dramatically, they are produced from similar progenitors. Understanding how this process is controlled could revolutionize our ability to treat reproductive disorders and infertility in humans. Evolution of Hermaphroditism: Sexual traits are among the most rapidly changing features of each species. To learn how these changes take place, and how developmental pathways constrain which ones occur, we are studying the evolution of mating systems in nematodes.

  • Jennifer Fischer, PhD

    Jennifer Fischer, PhD

    Associate Professor
    Science Center 128
    856 566-6919

    Medical schools employ a variety of curricula to educate students to be competent physicians. Currently we are evaluating the role and value of biomedical knowledge in clinical reasoning and diagnosing by obtaining feedback from practicing physicians. I also focus on understanding and utilizing educational technologies such as exam software, online learning management systems and virtual programs to enhance student learning.

  • Gary S. Goldberg, PhD

    Gary S. Goldberg, PhD

    Associate Professor
    Science Center B307
    856 566-6718

    Cells must communicate with each other to coordinate the development and survival of an animal. This communication can be mediated by diffusible factors that pass between cells, or by direct contact through cell junctions. I am interested in how intercellular communication affects cell growth and differentiation, with an emphasis on how cell communication can control tumor cell growth and prevent eye diseases.

  • Michael Henry, PhD

    Michael Henry, PhD

    Assistant Professor
    Science Center 320
    856 566-6970

    We use the yeast Saccharomyces cerevisiae as a model system to understand the molecular mechanisms by which RNA precursors are processed in the nucleus. More precisely, our goal is to understand the role of posttranslational protein modification in this process.

  • Ping Jiang, MD

    Ping Jiang, MD

    Assistant Professor
    Director, ES Cell and Transgenics Laboratory
    Department of Molecular Biology

    My laboratory, the ES Cell and Transgenics Laboratory at Rowan University School of Osteopathic Medicine, creates genetically modified mouse models for studies of human diseases and is open to the scientific communities in the United States as well as around the globe. Numerous transgenic, knockout and knockin mouse models were generated when I directed similar facilities at The Wistar Institute and Cold Spring Harbor Laboratory for over ten years.

  • Kai Mon Lee, PhD

    Kai Mon Lee, PhD

    Associate Professor
    Rowan Medicine Building, 2214
    856 566-6152

    We use 3D printing to fabricate three-dimensional (3D) scaffolds of varying architecture to provide 3D organization of neurons and their supporting cells with the objective of mimicking their 3D environment in the human brain. The scaffolds are constructed of biocompatible protein and protein composites. We use these platforms to study neurodegenerative diseases such as Alzheimer’s and Parkinson’s and, drug delivery. Another research interest of our lab is the fabrication of implantable protein-based neuron chips.

  • Eric Moss, PhD

    Eric Moss, PhD

    Associate Professor
    Science Center 312
    856 566-2896

    We study developmental timing, microRNAs and translational control in C. elegans and the mouse. The worm heterochronic gene lin-28 is regulated by microRNAs and encodes a specific mRNA-binding protein. Its human homologue, Lin28, appears also to be a microRNA-controlled developmental regulator.

  • Susan Muller-Weeks, PhD

    Susan Muller-Weeks, PhD

    Assistant Professor
    Science Center 130
    856 566-6097

    Research in the laboratory focuses on the repair of uracil in DNA, which is critical for the maintenance of genomic integrity. Specifically we are elucidating transcriptional and post-translational pathways that regulate expression of uracil-DNA glycosylase under normal cellular conditions and in response to anti-tumor agents.

  • Catherine L. Neary, PhD

    Assistant Professor
    Science Center
    856 566-6373

    Altered organelle function initiates a cell stress response; failure of this stress response results in the coordinated disassembly of the cell, known as apoptotic cell death. My interest is how metabolic stress affects the initiation of cell death, and how organelles, specifically the mitochondria and cytoskeleton, communicate during the death process. Additional projects in my laboratory include assessing the role of lactic acid metabolism in mitochondrial function, as well as cancer cell dependence on abnormal AMP-dependent kinase (AMPK) activation.

  • Randy Strich, PhD

    Randy Strich, PhD

    Science Center 354

    Our laboratory focuses on understanding how the transcription program is coupled to meiotic progression in budding yeast. A second project investigates the activity of the conserved C-type cyclin in directing the oxidative stress response and apoptosis in yeast and mammalian systems.

  • Brian P Wesier, PhD

    Brian P. Weiser, PhD

    Assistant Professor
    Science Center 307A

    My lab examines the architecture and function of protein-ligand and protein-protein complexes that are mediated by low-affinity interactions. We use chemical and molecular techniques to quantify and manipulate low-affinity interactions to examine their biochemical relevance. Current projects examine (1) DNA repair protein complexes and (2) neuronal deacylase enzymes.

We are pleased to announce a new program: Master of Science in Histopathology. In response to increased demand for highly competent technicians in translational research, we have developed this unique, hands-on program to prepare students to conduct biomedical research, including basic molecular and cell biology techniques, as well as the processing and analysis of primary tissue, in translational research. (read more!)

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